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Seminář Poulami Banik
Seminář Poulami Banik
“Characterization and targeted rescue of a novel PRPF31 mutation in retinitis pigmentosa” Retinitis pigmentosa (RP) is an inherited retinal disease characterized by photoreceptor loss. Mutations in the PRPF31 gene cause approximately 10% of cases of autosomal dominant RP. In this project, we characterize a novel intronic pathogenic variant in the PRPF31 gene. We provide evidence…
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BD Biosciences – Lunch and Learn Seminar
Dear colleagues, We would like to invite you to join Lunch & Learn seminar focused on Panel Design Strategy in Flow Cytometry. This session is designed to support anyone involved in planning, optimizing, or performing multicolor flow cytometry experiments by providing both conceptual guidance and practical demonstrations. More information and registration. Flyer Registration…
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Seminář Vladimír Varga
Seminář Vladimír Varga
“Towards understanding life cycle of Trypanosoma brucei” Trypanosoma brucei is a unicellular eukaryotic parasite of mammals causing sleeping sickness in humans. T. brucei is transmitted by the tsetse fly. In the fly trypanosomes migrate from the gut to salivary glands, which is associated with a defined sequence of changes to cells of the parasite. We…
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Seminář Diego André Florian Joseph
Seminář Diego André Florian Joseph
“The Fall of the Helicase of Dicer” Dicer is an RNase III endoribonuclease that processes pre-miRNAs and dsRNAs into small RNAs of ~21–28 nt, serving the miRNA and RNAi pathways. In mammals, Dicer’s ability to process long dsRNA is constrained by its helicase domain. Consequently, the miRNA pathway predominates, and RNAi is considered vestigial. This…
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Seminář – Srikant Ojha
Seminář – Srikant Ojha
“Plectin Loss Disrupts Mechanotransduction and Attenuates Hepatic Stellate Cell Activation” Liver fibrosis, a hallmark of fibroproliferative disorders that contribute to nearly 45% of global mortality, is primarily driven by the activation of hepatic stellate cells (HSCs). In response to injury-induced tissue stiffening, HSCs undergo mechanosignaling-dependent activation and deposit excessive extracellular matrix (ECM), perpetuating a self-sustaining…
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